<?xml version="1.0" encoding="UTF-8"?>
<?xml-stylesheet type="text/xsl" href="/sponet/themes/root/assets/xsl/rss.xsl"?>
<rss version="2.0" xmlns:opensearch="http://a9.com/-/spec/opensearch/1.1/" xmlns:atom="http://www.w3.org/2005/Atom" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:content="http://purl.org/rss/1.0/modules/content/" xmlns:slash="http://purl.org/rss/1.0/modules/slash/">
  <channel>
    <title>Ergebnis für Versionen - 3032933</title>
    <description>Treffer 1 - 2 von 2</description>
    <generator>Laminas_Feed_Writer 2 (https://getlaminas.org)</generator>
    <link>https://sponet.de/sponet/Search/Versions?sort=first_indexed+desc%2Cfirst_indexed+desc&amp;limit=50&amp;id=3032933&amp;search=versions&amp;lng=de</link>
    <opensearch:totalResults>2</opensearch:totalResults>
    <opensearch:startIndex>0</opensearch:startIndex>
    <opensearch:itemsPerPage>50</opensearch:itemsPerPage>
    <opensearch:Query role="request" searchTerms="3032933" startIndex="0"/>
    <atom:link rel="first" type="application/rss+xml" title="Zur ersten Seite springen" href="https://sponet.de/sponet/Search/Versions?sort=first_indexed+desc%2Cfirst_indexed+desc&amp;limit=50&amp;view=rss&amp;id=3032933&amp;search=versions&amp;lng=de"/>
    <atom:link rel="last" type="application/rss+xml" title="Zur letzten Seite springen" href="https://sponet.de/sponet/Search/Versions?sort=first_indexed+desc%2Cfirst_indexed+desc&amp;limit=50&amp;view=rss&amp;id=3032933&amp;search=versions&amp;lng=de&amp;page=1"/>
    <atom:link rel="self" type="application/rss+xml" href="https://sponet.de/sponet/Search/Versions?sort=first_indexed+desc%2Cfirst_indexed+desc&amp;limit=50&amp;view=rss&amp;id=3032933&amp;search=versions&amp;lng=de"/>
    <item>
      <title>Kardiorespiratorische Reaktionen auf Belastung unter akuter Hypoxie, Hyperoxie und Normoxie</title>
      <pubDate>Mon, 01 Jan 2001 03:28:25 +0100</pubDate>
      <link>https://sponet.de/sponet/Record/3032933</link>
      <guid>https://sponet.de/sponet/Record/3032933</guid>
      <author>Peltonen, J. E.</author>
      <author>Tikkanen, H. O.</author>
      <author>Rusko, H. K.</author>
      <dc:format>Artikel</dc:format>
      <dc:subject>Ausdauer</dc:subject>
      <dc:subject>Belastung</dc:subject>
      <dc:subject>Hypoxie</dc:subject>
      <dc:subject>Herz</dc:subject>
      <dc:subject>Kreislauf</dc:subject>
      <dc:subject>Atmung</dc:subject>
      <dc:format>Artikel</dc:format>
      <dc:creator>Peltonen, J. E.</dc:creator>
      <dc:creator>Tikkanen, H. O.</dc:creator>
      <dc:creator>Rusko, H. K.</dc:creator>
      <content:encoded><![CDATA[Es wird die Hypothese überprüft, dass eine akute Veränderung der Sauerstofffraktion in der Einatmungsluft (FIO2) keinen Einfluss auf das maximale Herzminutenvolumen hat, obwohl die VO2max und Leistung mit der FIO2 variieren. 6 Ausdauersportler unterzogen sich progressiver Fahrradergomterbelastung unter Hypoxie (FIO2=0.150), Normoxie (FIO2=0.209) und Hyperoxie (FIO2=0.320).  Wie erwartet sank die VO2max unter Hypoxie und stieg unter Hyperoxie im Vergleich zur Normoxie. Die maximale Wattleistung sank ebenfalls unter Hypoxie und tendierte zu einer Erhöhung unter Hyperoxie im Vergleich zur Normoxie. Im Gegensatz zur Hypothese betrug das maximale Herzminutenvolumen unter Hypoxie 25,99 l/min, 28,51 unter Normoxie und 30,13 unter Hyperoxie.]]></content:encoded>
      <slash:comments>0</slash:comments>
    </item>
    <item>
      <title>Kardiorespiratorische Reaktionen auf Belastung unter akuter Hypoxie, Hyperoxie und Normoxie</title>
      <pubDate>Mon, 01 Jan 2001 03:28:25 +0100</pubDate>
      <link>https://sponet.de/sponet/Record/4005486</link>
      <guid>https://sponet.de/sponet/Record/4005486</guid>
      <author>Peltonen, J. E.</author>
      <author>Tikkanen, H. O.</author>
      <author>Rusko, H. K.</author>
      <dc:format>Artikel</dc:format>
      <dc:subject>O2-Aufnahme</dc:subject>
      <dc:subject>Hypoxie</dc:subject>
      <dc:subject>Herzfrequenz</dc:subject>
      <dc:subject>Belastung</dc:subject>
      <dc:subject>Ausdauer</dc:subject>
      <dc:subject>Atmung</dc:subject>
      <dc:format>Artikel</dc:format>
      <dc:creator>Peltonen, J. E.</dc:creator>
      <dc:creator>Tikkanen, H. O.</dc:creator>
      <dc:creator>Rusko, H. K.</dc:creator>
      <content:encoded><![CDATA[There is a prevailing hypothesis that an acute change in the fraction of oxygen in inspired air (FIO2) has no effect on maximal cardiac output (), although maximal oxygen uptake () and exercise performance do vary along with FIO2. We tested this hypothesis in six endurance athletes during progressive cycle ergometer exercise in conditions of hypoxia (FIO2=0.150), normoxia (FIO2=0.209) and hyperoxia (FIO2=0.320). As expected,  decreased in hypoxia [mean (SD) 3.58 (0.45) l·min-1, P<0.05] and increased in hyperoxia [5.17 (0.34) l·min-1, P<0.05] in comparison with normoxia [4.55 (0.32) l·min-1]. Similarly, maximal power () decreased in hypoxia [334 (41) W, P<0.05] and tended to increase in hyperoxia [404 (58) W] in comparison with normoxia [383 (46) W]. Contrary to the hypothesis, was 25.99 (3.37) l·min-1 in hypoxia (P<0.05 compared to normoxia and hyperoxia), 28.51 (2.36) l·min-1 in normoxia and 30.13 (2.06) l·min-1 in hyperoxia. Our results can be interpreted to indicate that (1) the reduction in  in acute hypoxia is explained both by the narrowing of the arterio-venous oxygen difference and reduced , (2) reduced  in acute hypoxia may be beneficial by preventing a further decrease in pulmonary and peripheral oxygen diffusion, and (3) reduced  and  in acute hypoxia may be the result rather than the cause of the reduced  and skeletal muscle recruitment, thus supporting the existence of a central governor. ]]></content:encoded>
      <slash:comments>0</slash:comments>
    </item>
  </channel>
</rss>
