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    <item>
      <title>EPO-Konzentration und arterielle Hämoglobinsättigung bei supramaximaler Belastung</title>
      <pubDate>Fri, 01 Jan 1999 03:05:06 +0100</pubDate>
      <link>https://sponet.de/sponet/Record/4002298</link>
      <guid>https://sponet.de/sponet/Record/4002298</guid>
      <author>Roberts, D.</author>
      <author>Smith, D. J.</author>
      <dc:format>Artikel</dc:format>
      <dc:subject>EPO</dc:subject>
      <dc:subject>O2</dc:subject>
      <dc:subject>maximal</dc:subject>
      <dc:subject>Blut</dc:subject>
      <dc:subject>Belastung</dc:subject>
      <dc:subject>Ausdauer</dc:subject>
      <dc:format>Artikel</dc:format>
      <dc:creator>Roberts, D.</dc:creator>
      <dc:creator>Smith, D. J.</dc:creator>
      <content:encoded><![CDATA[The aim of this study was to determine if the hypoxaemic stimulus generated by intense exercise results in the physiological response of increased erythropoietin production. Twenty athletes exercised for 3 min at 109 +/- 2.8% (mean +/-s) maximal oxygen consumption. Estimated oxyhaemoglobin saturation was measured by reflective probe pulse oximetry (Nellcor N200) and was validated against arterial oxyhaemoglobin saturation by CO-oximetry in eight athletes. Serum erythropoietin concentrations - as measured using the INCSTAR Epo-Trac radioimmunoassay - increased significantly by 28 +/- 9% at 24 h post-exercise in 11 participants, who also had an arterial oxyhaemoglobin saturation 91% (P < 0.05). Decreased ferritin levels and increased reticulocyte counts were observed at 96 h post-exercise. However, no significant changes in erythropoietin levels were observed in nine non-desaturating athletes and eight non-exercise controls. Good agreement was shown between arterial oxyhaemoglobin saturation and percent estimated oxyhaemoglobin saturation (limits of agreement = -3.9 to 3.7%). In conclusion, short supramaximal exercise can induce both hypoxaemia and increased erythropoietin levels in well-trained  individuals. The decline of arterial hypoxaemia levels below 91% during exercise appears to be necessary for the exercise-induced elevation of serum erythropoietin levels. Furthermore, reflective probe pulse oximetry  was found to be a valid predictor of percent arterial oxyhaemoglobin saturation during supramaximal exercise when percent estimated oxyhaemoglobin saturation >_86%.]]></content:encoded>
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    <item>
      <title>Erythropoietinkonzentration arterielle Hämoglobinsättigung bei supramaximaler Belastung</title>
      <pubDate>Fri, 01 Jan 1999 03:05:06 +0100</pubDate>
      <link>https://sponet.de/sponet/Record/3030207</link>
      <guid>https://sponet.de/sponet/Record/3030207</guid>
      <author>Roberts, D.</author>
      <author>Smith, D. J.</author>
      <dc:format>Artikel</dc:format>
      <dc:subject>Hypoxie</dc:subject>
      <dc:subject>aerob</dc:subject>
      <dc:subject>Belastung</dc:subject>
      <dc:subject>maximal</dc:subject>
      <dc:subject>Blut</dc:subject>
      <dc:subject>O2</dc:subject>
      <dc:subject>EPO</dc:subject>
      <dc:format>Artikel</dc:format>
      <dc:creator>Roberts, D.</dc:creator>
      <dc:creator>Smith, D. J.</dc:creator>
      <content:encoded><![CDATA[Zielstellung:
Untersuchung ob ein hypoxämischer Stimulus durch intensive Belastung eine erhöhte EPO-Produktion bewirkt.
20 Sportler (7 Bergsteiger, 11 Volleyballspieler, 2 Eisschnelläufer) absolvierten in 1000m Höhe über 3 min bei 109+/-2.8% VO2max einen Fahrrardergometertest. 
Ergebnis: Kurze supramaximale Belastung kann sowohl eine arterielle Hypoxie als auch einen erhöhen EPO-Spiegel bei guttrainierten Personen bewirken. Der Abfall des arteriellen Hypoxiespiegels unter 91% während Belastung scheint für die belastungsbedingte Erhöhung von Serum-_EPO notwendig zu sein.]]></content:encoded>
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